COVID jabs form spike proteins in heart cells, causing irregular heart contractions and full blown cardiomyopathy, researchers find – NaturalNews.com
COVID jabs form spike proteins in heart cells, causing irregular heart contractions and full blown cardiomyopathy, researchers find
Researchers find mechanisms of injury behind COVID jabs
Within 48 hours of vaccination, the COVID-19 mRNA vaccines are designed to transcribe a specific sequence of coronavirus spike protein in the deltoid muscle of vaccine recipients. The mRNA transcription process is intended to hijack protein synthesis of human cells at the vaccination site, to ultimately summon immune responsive cells. The foreign spike proteins are supposed to be neutralized in the deltoid muscle, leaving behind an enduring immune response. This was the promise of vaccine makers, but scientists are finding out that the transcription process and the neutralization of spike proteins does not work as advertised, and the transcription process can even harm vital organs. Not only is the immune response insufficient with these vaccines, but the spike proteins can also be transcribed in heart cells, causing serious and life-threatening risks to the vaccine recipient.
In the study – Cardiac side effects of RNA-based SARS-CoV-2 vaccines: Hidden cardio-toxic effects of mRNA-1273 and BNT162b2 on ventricular myocyte function and structure – researchers found something very troubling about the current mRNA medical experiment, something that may have repercussions for human health for many years to come.
In just 48 hours, both the Pfizer and Moderna covid vaccines caused distinct cell abnormalities, suggesting that the body may reject synthetic mRNA synthesis and have a toxicity reaction within the cells. “The findings support both the diagnosis and treatment of cardiac events following mRNA-based COVID vaccination,” the authors wrote. They also said the findings help explain the persistent cardiac symptoms that are observed among long-COVID patients. Most long-COVID patients have been vaccinated with Pfizer and Moderna jabs.
The standard dosage for the Moderna jab is 100 micrograms and dosage for Pfizer is 30 micrograms. The study authors introduced the mRNA vaccines to cell cultures from rat and human heart cells and administered both the Pfizer and Moderna at 100 micrograms. Within 48 hours, the heart cells were replicating spike proteins, causing abnormal heart contractions.
Heart cells inundated with Pfizer’s spike proteins showed increased protein kinase A (PKA) activity and stronger heart contractions. Heart cells inundated with Moderna’s spike proteins had disrupted calcium regulation and developed irregular arrhythmic and irregular peristaltic heart contractions. The researchers traced the damage back to disturbances among the RyR2 receptors, which coordinate heart contractions using calcium.
COVID jabs cause more than myocarditis: They also cause cardiomyopathy
The researchers compared normal contractions in unvaccinated rat heart cells with the abnormal contractions found in vaccinated heart cells. The researchers concluded that the COVID jabs cause damage at the cellular level, damage that aligns closer with cardiomyopathy than it does with myocarditis. In myocarditis, the heart tissue is inflamed. In cardiomyopathy, the heart muscles suffer toxic stress and become both structurally and functionally abnormal in the absence of other heart diseases. The newly-discovered pathology of cardiomyopathy explains why young, previously healthy people are dying post-vaccination. This evidence could prove vital for future court cases against the covid-19 vaccine makers. The research paper was expedited to a rapid communication paper and was published more quickly than a standard research article because the scientists believe that the findings need to be shared immediately with the academic community.
“The present preclinical cardiac safety data point to the need for a reassessment of the risk–benefit ratio of RNA-based SARS-Cov-2 vaccines, given indication of their cardiotoxicity,” the authors wrote. The heart abnormalities exhibited in the cells, likely caused by disruption to RyR2 and increased PKA protein levels, “are risk factors for sudden cardiac death, ventricular tachyarrhythmias and contractile dysfunction,” they added.
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