Why Antiviral Pharmaceuticals Can’t Possibly Work as Claimed

My recent articles focused on the fantasy of the spike protein:

See here: https://anitabaxasmd.substack.com/p/gas-lighting-and-misdirection-with

and here: https://anitabaxasmd.substack.com/p/misleading-criticism-about-the-dangers

I’m following up with this post about the illusion of antiviral pharmaceuticals supposedly stopping the replication of viruses. For one thing, there is no evidence, that viruses exist, see here:

https://anitabaxasmd.substack.com/p/the-questionable-virus-theory

I delved into claims of Pfizer regarding their antiviral drug Nirmatrelvir, better known as Paxlovid™.
On their website for health professionals ¹ they write that it works by inhibiting a certain viral protein, Mpro, necessary for a virus to multiply. They claim that “Nirmatrelvir exhibited antiviral activity against SARS-CoV-2 in cell culture studies across the Alpha, Beta, Gamma, Delta, Lambda, Mu, and Omicron variants.”

One of their citations² says: “The genome of SARS-CoV-2 encodes non-structural proteins [SARS-CoV-2 Mpro (main protease), also known as 3-chymotrypsin-like cysteine protease (CCP or 3CLpro), papain-like protease, and RNA-dependent RNA polymerase (RdRp)], helicase, structural proteins (spike glycoprotein), and accessory proteins. The non-structural proteins play a key role during the virus’s life cycle, and spike glycoprotein is necessary for the interactions of the virus with the host cell receptors during viral entry. The non-structural and structural proteins were recognized as promising targets for the design and development of antiviral agents against SARS and MERS.”

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